
Sandra Lee Petersen SLP
Speech-Language Pathologist
805 W STONEMILL AVE ADDISON IL, 60101About
Dr. Sandra Petersen is a speech language pathologist practicing in ADDISON, IL. Dr. Petersen specializes in speech, language and swallowing disorders in patients. As a speech language pathologist, Dr. Petersen evaluates, diagnoses and treats patients with communication and swallowing troubles. These conditions may be due to developmental delay, brain injury, hearing loss, autism, stroke or other diseases and injuries. Dr. Petersen helps patients make sounds and improve their voices through various methods. Speech language pathologists also work with patients to strengthen muscles used to speak and swallow, and work with individuals and families to help cope with their conditions.
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Expert Publications
Data provided by the National Library of Medicine- Evidence that GABAergic neurons in the preoptic area of the rat brain are targets of 2,3,7,8-tetrachlorodibenzo-p-dioxin during development.
- Increased concentrations of radioisotopically-labeled complementary ribonucleic acid probe, dextran sulfate, and dithiothreitol in the hybridization buffer can improve results of in situ hybridization histochemistry.
- Glutamatergic signaling through the N-methyl-D-aspartate receptor directly activates medial subpopulations of luteinizing hormone-releasing hormone (LHRH) neurons, but does not appear to mediate the effects of estradiol on LHRH gene expression.
- The distribution of progestin receptor mRNA in rat brainstem.
- Direct and indirect regulation of gonadotropin-releasing hormone neurons by estradiol.
- Estrogen receptor-beta in oxytocin and vasopressin neurons of the rat and human hypothalamus: Immunocytochemical and in situ hybridization studies.
- Dual-phenotype GABA/glutamate neurons in adult preoptic area: sexual dimorphism and function.
- Regulation of gonadotropin-releasing hormone secretion by cannabinoids.
- The aryl hydrocarbon receptor pathway and sexual differentiation of neuroendocrine functions.
- The gad2 promoter is a transcriptional target of estrogen receptor (ER)alpha and ER beta: a unifying hypothesis to explain diverse effects of estradiol.
- Central role of TRAF-interacting protein in a new model of brain sexual differentiation.
- Identification of prolactin-sensitive GABA and kisspeptin neurons in regions of the rat hypothalamus involved in the control of fertility.
- Aryl hydrocarbon receptor activation in lactotropes and gonadotropes interferes with estradiol-dependent and -independent preprolactin, glycoprotein alpha and luteinizing hormone beta gene expression.
- Estradiol acts through nuclear- and membrane-initiated mechanisms to maintain a balance between GABAergic and glutamatergic signaling in the brain: implications for hormone replacement therapy.
- Frequency-dependent recruitment of fast amino acid and slow neuropeptide neurotransmitter release controls gonadotropin-releasing hormone neuron excitability.
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