
Dr. Joel David Taurog MD
Rheumatologist | Rheumatology
5323 Harry Hines Blvd Dallas TX, 75390About
Dr. Taurog has been a faculty member in the Division of Rheumatic Diseases at UT Southwestern since 1986. Since joining the faculty he has served as attending physician in rheumatology and internal me ...
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Rheumatology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Sharing of an HLA-B27-restricted H-Y antigen between rat and mouse.
- A high incidence of Shigella-induced arthritis in a primate species: major histocompatibility complex class I molecules associated with resistance and susceptibility, and their relationship to HLA-B27.
- HLA-B27 has no effect on the phenotypic expression of progressive ankylosis in ank/ank mice.
- Development of spontaneous arthritis in beta2-microglobulin-deficient mice without expression of HLA-B27: association with deficiency of endogenous major histocompatibility complex class I expression.
- HLA-DR4 and the spondyloarthropathies.
- CD8 alpha beta T cells are not essential to the pathogenesis of arthritis or colitis in HLA-B27 transgenic rats.
- In vitro mutagenesis of HLA-B27: single and multiple amino acid substitutions at consensus B27 sites identify distinct monoclonal antibody-defined epitopes.
- Formation of HLA-B27 homodimers and their relationship to assembly kinetics.
- HLA-B27 in transgenic rats forms disulfide-linked heavy chain oligomers and multimers that bind to the chaperone BiP.
- Morris Ziff, PhD, MD, 1913-2005.
- Adoptive transfer of nontransgenic mesenteric lymph node cells induces colitis in athymic HLA-B27 transgenic nude rats.
- Additional human beta2-microglobulin curbs HLA-B27 misfolding and promotes arthritis and spondylitis without colitis in male HLA-B27-transgenic rats.
- In vitro mutagenesis of HLA-B27. Amino acid substitutions at position 67 disrupt anti-B27 monoclonal antibody binding in direct relation to the size of the substituted side chain.
- Mechanisms of disruption of the articular cartilage surface in inflammation. Neutrophil elastase increases availability of collagen type II epitopes for binding with antibody on the surface of articular cartilage.
- The mystery of HLA-B27: if it isn't one thing, it's another.
Awards
- 2013 Master of the American College of Rheumatology
- Glenn Clark Lectureship - 2008 University of Tennessee, Memphis
- 1997 William M. & Gatha Burnett Professorship for Arthritis Research - UT
- Kroc Lectureship - 1993 University of Michigan
- 1992 Carol-Nachman Prize for Rheumatology - State Capital of Wiesbaden, Ge
- 1991 Rheumatologist of the Year - Abstract Rhumato (French Journal)
Professional Memberships
- Member American Association of Immunologists
- Member American College of Rheumatology
- Member American Society for Clinical Investigation
Fellowships
- National Institutes of Health-Research, Immunology/rheumatology 1980
Dr. Joel David Taurog MD's Practice location
Practice At 5323 Harry Hines Blvd
5323 Harry Hines Blvd -Dallas, TX 75390Get Direction
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Nearby Providers
- Dr. Guillermo Andres Quiceno M.D.5323 HARRY HINES BLVD. DALLAS TX 75390
- Dr. Don E Cheatum MD8440 Walnut Hill Ln Dallas TX 75231
- Dr. Uzma A Syeda MD210 N. Park Blvd, Grapevine TX 76051
- Dr. Pooja Banerjee MD8230 Walnut Hill Ln Dallas TX 75231
- Dr. Claire A. Horn MD5323 Harry Hines Blvd. Dallas TX 75390
- Dr. Richard Leo Stern M.D.12740 Hillcrest Rd Dallas TX 75230
Nearest Hospitals
UT SOUTHWESTERN UNIVERSITY HOSPITAL-ZALE LIPSHYl
5151 HARRY HINES BLVD DALLAS TX 75390PARKLAND HEALTH AND HOSPITAL SYSTEMl
5200 HARRY HINES BLVD DALLAS TX 75235