
Dr. Faye S Silverstein MD
Neurologist | Neurology with Special Qualifications in Child Neurology
1500 East Medical Cneter Dr 1st Floor Taubman Ct Ann Arbor MI, 48109About
Dr. Faye Silverstein is a distinguished Neurologist in Ann Arbor, MI. Dr. Silverstein specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Silverstein employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Silverstein is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Mc Gill Univ- Fac Of Med- Montreal- Que- Canada 1975
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Tat, a human immunodeficiency virus-1-derived protein, augments excitotoxic hippocampal injury in neonatal rats.
- Expanding the scope of child neurology for the 21st century.
- Inhibition of TNF-alpha can attenuate or exacerbate excitotoxic injury in neonatal rat brain.
- Experimental gliosarcoma induces chemokine receptor expression in rat brain.
- Acute excitotoxic injury induces expression of monocyte chemoattractant protein-1 and its receptor, CCR2, in neonatal rat brain.
- Monocyte chemoattractant protein-1 is a mediator of acute excitotoxic injury in neonatal rat brain.
- Hypoxic-ischemic injury results in acute disruption of myelin gene expression and death of oligodendroglial precursors in neonatal mice.
- Attenuation of hypoxia-ischemia-induced monocyte chemoattractant protein-1 expression in brain of neonatal mice deficient in interleukin-1 converting enzyme.
- Successful use of rt-PA in pediatric stroke.
- AMPA-Induced suppression of oligodendroglial gene expression in neonatal rat brain.
- Hypoxic-ischemic injury induces macrophage inflammatory protein-1alpha expression in immature rat brain.
- N-methyl-D-aspartate-mediated injury enhances quisqualic acid-stimulated phosphoinositide turnover in perinatal rats.
- Excitatory amino acids contribute to the pathogenesis of perinatal hypoxic-ischemic brain injury.
- Complement activation contributes to hypoxic-ischemic brain injury in neonatal rats.
- The microsomal calcium-ATPase inhibitor thapsigargin is a neurotoxin in perinatal rodent brain.
Dr. Faye S Silverstein MD's Practice location
Practice At 1500 East Medical Cneter Dr 1st Floor Taubman Ctr Recp D
1500 East Medical Cneter Dr 1st Floor Taubman Ct -Ann Arbor, MI 48109Get Direction
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