Sandra K Laudon MS, LCPC, QMHP
Counselor/Therapist | Mental Health
325 IL ROUTE 2 DIXON IL, 61021About
Sandra Laudon is an Addiction Medicine Physician in DIXON, IL. Sandra evaluates patients using many different procedures, in order to determine what treatments must be carried out in order to properly assess their symptoms. Addiction Medicine Physicians provide medical care in addition to consultation for each patient and their families.
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Expert Publications
Data provided by the National Library of Medicine- Myocardin is a key regulator of CArG-dependent transcription of multiple smooth muscle marker genes.
- Smooth muscle alpha-actin gene requires two E-boxes for proper expression in vivo and is a target of class I basic helix-loop-helix proteins.
- Combinatorial control of smooth muscle-specific gene expression.
- Platelet-derived growth factor-BB and Ets-1 transcription factor negatively regulate transcription of multiple smooth muscle cell differentiation marker genes.
- Myocardin and Prx1 contribute to angiotensin II-induced expression of smooth muscle alpha-actin.
- Lost in transdifferentiation.
- Forced expression of myocardin is not sufficient for induction of smooth muscle differentiation in multipotential embryonic cells.
- Molecular regulation of vascular smooth muscle cell differentiation in development and disease.
- Transforming growth factor-beta1 signaling contributes to development of smooth muscle cells from embryonic stem cells.
- ANG II type 2 receptor regulates smooth muscle growth and force generation in late fetal mouse development.
- Kruppel-like factor 4 abrogates myocardin-induced activation of smooth muscle gene expression.
- 5' CArG degeneracy in smooth muscle alpha-actin is required for injury-induced gene suppression in vivo.
- Molecular determinants of vascular smooth muscle cell diversity.
- Regulation of alpha-smooth muscle actin expression in granulation tissue myofibroblasts is dependent on the intronic CArG element and the transforming growth factor-beta1 control element.
- PIAS1 activates the expression of smooth muscle cell differentiation marker genes by interacting with serum response factor and class I basic helix-loop-helix proteins.
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