Dr. Ana M. Ibrado M.D.
Emergency Physician
1150 Varnum St. N. E. Providence Hospital Washington DC, 20017Rating
5/5
About
Dr. Ana Ibrado practices Emergency Medicine in Washington, DC. Dr. Ibrado assesses patients who seek immediate medical attention at any time of day or night. Emergency Medicine Physicians are trained to efficiently work with each patient and situation no matter how acute or life-threatening. Dr. Ibrado examines patients, determines means of testing, diagnoses conditions, and decides the best treatment methods.
Education and Training
Medical University of South Carolina College of Medicine 1998
Board Certification
Emergency MedicineAmerican Board of Emergency MedicineABEM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Phase I and pharmacologic study of a 3-hour infusion of paclitaxel followed by cisplatinum and 5-fluorouracil in patients with advanced solid tumors.
- Granulocyte-macrophage colony-stimulating factor/interleukin-3 fusion protein (pIXY 321) enhances high-dose Ara-C-induced programmed cell death or apoptosis in human myeloid leukemia cells.
- Deoxycytidine protects normal bone marrow progenitors against Ara-C and gemcitabine cytotoxicity without compromising their activity against cisplatin-resistant human ovarian cancer cells.
- Effect of combined treatment with interleukin-3 and interleukin-6 on 4-hydroperoxycyclophosphamide-mediated reduction of glutathione levels and cytotoxicity in normal and leukemic bone marrow progenitor cells.
- Treatment with interleukin-3 plus granulocyte-macrophage colony-stimulating factors improves the selectivity of Ara-C in vitro against acute myeloid leukemia blasts.
- pIXY321 protects against Ara-C or taxol-induced apoptosis and loss of clonogenic survival of normal human bone marrow progenitor cells.
- Effect of hemopoietic growth factors G-CSF and pIXY 321 on the activity of high dose Ara-C in human myeloid leukemia cells.
- Combined antileukemic activity of pIXY 321 and Ara-C against human acute myeloid leukemia cells.
- Characterization of a human myeloid leukemia cell line highly resistant to taxol.
- 1-beta-D-arabinofuranosylcytosine-, mitoxantrone-, and paclitaxel-induced apoptosis in HL-60 cells: improved method for detection of internucleosomal DNA fragmentation.
- Taxol induces internucleosomal DNA fragmentation associated with programmed cell death in human myeloid leukemia cells.
- High-dose mitoxantrone induces programmed cell death or apoptosis in human myeloid leukemia cells.
- Effect of combined treatment with interleukin-3 and interleukin-6 on 4-hydroperoxycyclo-phosphamide-induced programmed cell death or apoptosis in human myeloid leukemia cells.
- Overexpression of Bcl-2 or Bcl-xL inhibits Ara-C-induced CPP32/Yama protease
- Bcl-xL overexpression inhibits taxol-induced Yama protease activity and apoptosis.
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