Rosalie M Uht
Pathologist | Anatomic Pathology & Clinical Pathology
Lee St Charlottesville VA, 22908About
Dr. Rosalie Uht is a pathologist practicing in Charlottesville, VA. Dr. Uht is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Uht can help your primary care doctor make a diagnosis about your medical condition. Dr. Uht may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Board Certification
PathologyAmerican Board of PathologyABP- Neuropathology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Estrogen receptor pathways to AP-1.
- Regulation of KLF5 involves the Sp1 transcription factor in human epithelial cells.
- A conserved lysine in the estrogen receptor DNA binding domain regulates ligand activation profiles at AP-1 sites, possibly by controlling interactions with a modulating repressor.
- Estrogen receptor (ER)beta isoforms rather than ERalpha regulate corticotropin-releasing hormone promoter activity through an alternate pathway.
- The protein kinase C-eta isoform induces proliferation in glioblastoma cell lines through an ERK/Elk-1 pathway.
- Estradiol regulates corticotropin-releasing hormone gene (crh) expression in a rapid and phasic manner that parallels estrogen receptor-alpha and -beta recruitment to a 3',5'-cyclic adenosine 5'-monophosphate regulatory region of the proximal crh pro
- Histone deacetylase 1 (HDAC1) participates in the down-regulation of corticotropin releasing hormone gene (crh) expression.
- Glucocorticoids are Required for Food Deprivation-Induced Increases in Hypothalamic Neuropeptide Y Expression.
- The ERβ ligand 5α-androstane, 3β,17β-diol (3β-diol) regulates hypothalamic oxytocin (Oxt) gene expression.
- Estrogen receptors and the regulation of neural stress responses.
- The androgen metabolite, 5α-androstane-3β,17β-diol (3β-diol), activates the oxytocin promoter through an estrogen receptor-β pathway.
- Hypothalamic and amygdalar cell lines differ markedly in mitochondrial rather than nuclear encoded gene expression.
- Mechanisms by Which 17β-Estradiol (E2) Suppress Neuronal cox-2 Gene Expression.
- CpG methylation and the methyl CpG binding protein 2 (MeCP2) are required for restraining corticotropin releasing hormone (CRH) gene expression.
- Estradiol (E2)- and tamoxifen (Tmx)-bound ER-alpha (ERα) interact differentially with histone deacetylases 1 and 3 (HDACs 1 and 3).
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Nearby Providers
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Mark R. Wick OtherLEE ST CHARLOTTESVILLE VA 22908![]()
Dr. James D. Gorham M.D. PH.D.1215 LEE ST CHARLOTTESVILLE VA 22908![]()
Dr. Thomas D Bourne MDUva Hospital Charlottesville VA 22908![]()
Dr. Mani S. Mahadevan M.D.3rd Hospital Dr Charlottesville VA 22908![]()
Dr. Yasmin I Lutterbie MD1215 Lee St Charlottesville VA 22908![]()
Dr. Alejandro A Gru MD1215 LEE ST CHARLOTTESVILLE VA 22908
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UNIVERSITY OF VIRGINIA MEDICAL CENTERl
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