Christine Edry Seidman MD
Cardiologist | Cardiovascular Disease
77 Avenue Louis Pasteur Nrb Harvard Medical Scho Boston MA, 02115Rating
5/5
About
Dr. Christine Seidman is a cardiologist practicing in Boston, MA. Dr. Seidman specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Seidman also practices preventative medicine, helping patients maintain a heart-healthy life.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Sarcomere protein gene mutations in hypertrophic cardiomyopathy of the elderly.
- Accelerated cardiomyopathy in mice with overexpression of cardiac G(s)alpha and a missense mutation in the alpha-myosin heavy chain.
- Constitutively active AMP kinase mutations cause glycogen storage disease mimicking hypertrophic cardiomyopathy.
- The L-type calcium channel inhibitor diltiazem prevents cardiomyopathy in a mouse model.
- Assessment of diastolic function with Doppler tissue imaging to predict genotype in preclinical hypertrophic cardiomyopathy.
- Contemporary evaluation and management of hypertrophic cardiomyopathy.
- Phenotypic diversity in hypertrophic cardiomyopathy.
- Dilated cardiomyopathy and heart failure caused by a mutation in phospholamban.
- Tbx5 is required for forelimb bud formation and continued outgrowth.
- Transgenic mice overexpressing mutant PRKAG2 define the cause of Wolff-Parkinson-White syndrome in glycogen storage cardiomyopathy.
- Consequences of pressure overload on sarcomere protein mutation-induced hypertrophic cardiomyopathy.
- Electrophysiologic characterization and postnatal development of ventricular pre-excitation in a mouse model of cardiac hypertrophy and Wolff-Parkinson-White syndrome.
- A novel missense mutation in the paired domain of PAX9 causes non-syndromic oligodontia.
- Differential cross-bridge kinetics of FHC myosin mutations R403Q and R453C in heterozygous mouse myocardium.
- Role of cardiac myosin binding protein C in sustaining left ventricular systolic stiffening.
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